The trigger finger
The trigger finger is a rather widespread phenomenon from an epidemiological point of view and relatively annoying.
The mechanism underlying this disorder is called stenosing tenosynovitis.
Ie it is an inflammation of the tendon sheath of the flexor tendon of a finger resulting in the formation of a nodule at the level of the tendon.
The inflammation of the sheath and at the same time the presence of a nodule tendon affects the possibility of sliding of the tendon in its sheath.
It therefore establishes a mechanical discomfort that occurs initially with a feeling of crackle during the movement and in the more advanced stages with a real impediment to flow.
If the size of the nodule is not excessive,, the finger can be flexed with a grater muscular effort but the abrupt transition of the nodule through the pulley of the tendon causes a twitchy advancement of the flexor.
Frequently the onset of a trigger finger can have a mechanical base.
Dynamic alteration of the joints of the upper limb can lead the tendons of the fingers to work according to the physiological axes, forcing some structures to excessive efforts at the expense of others.
A trigger finger may, however, be also an expression of a discomfort of the autonomous neuro- vegetative systems, that regulate the involuntary functions.
Among them, for example the size of the caliber of the vessels, sweating, tissue tropism, hydration, the distribution of body heat, and many more.
The Osteopath intervenes by balancing both the mechanical and the neuro-vegetative functionality.
Moreover, the trigger finger is a quite dynamic phenomenon, ie it is easy to see how this disorder can persist for months and then regress independently without any intervention.
In fact in these cases, despite the regression of the symptom, the basic osteopathic framework remains active and new nodular formations tend to form later on, perhaps in different positions compared to the earlier ones, then following the same trend.
Such patients usually present a chronic trigger finger.
The solution proposed and implemented according to osteopathy is based on a global rebalancing of the patient from the primary lesion outbreaks.
As an example we can consider the case of an employee with a trigger finger on the right thumb.
This patient previously had been operated for a similar problem to a finger of the contralateral hand.
Osteopathic examination showed that the patient had a high level of compression skull with a distorted dynamic at the level of the right zygomatic bone and of the right temporo-zygomatic joint.
There were other restrictions on the right ulna and on the fifth dorsal vertebra adapted in rotation and tilt to the left.
Incidentally, the fingers of the hand did not show any primary lesion.
I treated this patient for several months and I checked the situation at a later stage.
I can consider to have solved the problem in a stable manner without the need for further intervention.
In fact, in these cases the possibility of local intervention, also not osteopathic, is not excluded but it is perfectly useless to eliminate an effect without first tackling the causes that have generated it.
Therefore, in the case of trigger finger, an osteopathic revision is absolutely recommended.